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My pet idea is that rather than targetting cancer, we should be trying to find and correct DNA-errors before they become cancerous. Otherwise we are just playing whack-a-mole with a slowly degenerating cell population.


Most cells that divide will eventually have DNA errors over time, but will not always become cancerous.

If we could find and repair DNA errors in normal cells, we could do it in cancerous cells as well. I don't think there's much of a difference.




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